| |
Crufts & Shows |
|
|
you can view this email in brows
you can view this email
in browser click here
|
|
|
|
|
|
|
|
|
|
|
Dear Breeder
We are pleased to announce that LABOKLIN will be at Crufts from
6th to 9th March 2025 and we look forward to seeing you there.
Our stand is located in Hall 3 opposite the restaurant, it is stand number 3-20 as shown in the diagram.
Meet the team
We are pleased to announce that
our genetic expert Dr Christoph
Beiztinger will be at Crufts and
will be pleased to answer any
questions you may have.
We always talk on the phone, by email or even via facebook, however, at Crufts we are very excited to meet you in person, please stop by and say hi.
10% discount on all DNA tests submitted at Crufts !
and this offer includes our Breed Specific DNA Bundles
and the brand new KC breed bundles
How to submit a sample at Crufts:
How to submit a sample at Crufts :
- Bring your dog to our stand 3-20: we will take a DNA sample for your genetic test, all you need to do is complete the order form and pay the fees. Or,
- If you don't want to wait in the queue, you can prepare your sample in advance and bring it together with the attached order form with you to our stand. You can order a free DNA testing kit on our website, click on the following link to order:
Request sample materials
We will send you a testing kit which also contains order form and instructions on how to take DNA sample from the mouth. Prepare your sample up to a week before your planned visit, just hand the sample to us at the show.
- If you prefer to use blood for your test, ask your vet to collect 0.5-1 ml of whole blood in EDTA blood tube, bring it together with the completed order form to the show, just hand it to us. (you can order free EDTA blood tube from our website http://www.laboklin.co.uk )
Payments can be made by card, cheque, cash, postal Order, bank transfer and paypal. Turnaround: 1-3 weeks for the majority of tests from Monday 10th March. The attached order form will only be accepted at Crufts 2025. If you have any questions, please email info@laboklin.co.uk or ring 0161 2823066. We look forward to seeing you at Crufts.
Kind regards
The Laboklin Team LABOKLIN (UK)
Laboratory For Veterinary
Diagnostics
Unit 20, Wheel Forge Way
Trafford Park
Manchester
M17 1EH
Tel: 0161 282 3066
www.laboklin.co.uk
www.facebook.com/Laboklin
|
|
new
Dog Genetics XXL DNA Bundle
Discover Your Dog's Genetic Makeup with the New Dog Genetics XXL Dog Bundle
Unveil the secrets of your dog's DNA with the comprehensive Dog Genetics XXL Dog Bundle, which analyzes over
340 genetic variants to provide you with detailed insights into your dog's health, characteristics,
and genetic makeup. This all-inclusive test is perfect for both purebred dogs and mixed breeds.
What You Will Receive:
- Hereditary Diseases: Identify genetic health risks that could affect your dog.
- Genetic Risk Factors: Understand potential health threats and take preventive measures.
- Coat Colors and Characteristics: Discover the genetic factors behind your dog's unique
appearance.
Who Can Benefit?
- Breeders: Make informed breeding decisions to enhance the genetic health of your breed and
ensure healthier puppies.
- Dog Owners: Gain a deeper understanding of your dog's genetics to uncover potential health
risks and ensure their well-being.
- Veterinarians: Enhance your diagnostic and treatment plans with valuable genetic data for
better patient care.
- Breeding Clubs: Equip your members with advanced genetic testing to support responsible and
informed breeding practices.
The Dog Genetics XXL Dog Bundle is designed to cater to everyone from dog enthusiasts and breeders to veterinarians
and breeding clubsâ??providing unparalleled genetic insights to help you care for your canine companions better.
Our Canine XXL DNA bundle is a large collection of genetic tests designed to unlock the secrets of your dog's health and other traits. This extraordinary bundle combines
a diverse range of tests carefully selected to reveal fascinating insights about your dog's genetic makeup and genetic predispositions.
And the best part? It is available at a great price.
The Dog Genetics XXL Dog Bundle is performed using the new Next Generation Sequencing (NGS) technology.
Dog Genetics XXL Dog Bundle you with extensive information on genetic diseases and risk factors as well as on coat colours and traits.
cost £ 132.00
more information is available on the following link:
Dog Genetics XXL Dog Bundle
|
new
Lysosomal Storage Diseases (LSD)
Lysosomal Storage Diseases (LSD)
Lysosomal storage diseases refer to a collection of disorders characterized by the body's inability to produce essential metabolic enzymes. Lysosomes serve as cellular 'recycling centres' converting worn-out materials into substances that cells can utilize. When specific metabolic enzymes are lacking, the unprocessed material accumulates, leading to tissue damage and eventual failure of organ systems.
Symptoms include decreased vision, changes in behaviour, ataxia, incoordination, lethargy, weakness, tremors, seizures, stiff gait and dementia.
Please note that the variant which cause the disease in Dalmatian is different from the variant which causes it in Doberman and so the test is also different. We will run the test that is relevant to the breed. If the breed isn't listed, please specify which variant(s) you would like us to perform. Each variant will be charged separatedly.
additional information
In Dalmatian, a genetic variant in the CNP gene is responsible for lysosomal storage disease. Affected dogs exhibit gradually worsening symptoms such as abnormal behavior, cognitive decline, loss of coordination, apparent visual impairment, anxiety, pacing and circling, hypersensitivity, sleep disturbances, and loss of control over urination and defecation. These symptoms typically begin to appear around 18 months of age.
In Doberman dogs, a genetic variant in the MAN2B1 gene causes lysosomal storage disease. Symptoms in affected dogs can be observed as early as 2 months old, including clumsiness, difficulty standing, and frequent falls. Neurological examinations reveal slightly reduced mental alertness, proprioceptive ataxia, asymmetric reduced menace response, and moderate divergent strabismus.
Note: different variant affects each breed
cost 48.00
more information is available on the following link:
Lysosomal Storage Diseases (LSD)
|
new
Paradoxical Pseudomyotonia (PP)
Paradoxical Pseudomyotonia is a hereditary condition that affects English Cocker Spaniels and English Springer Spaniels.
The disease is characterized by episodes of muscle stiffness that are triggered by exercise. These episodes occur suddenly during exercise and cause a generalized myotonic-like muscle stiffness.
Symptoms typically manifest between the ages of 3 months and 2 years. During these episodes, muscles take longer to relax after voluntary contraction and remain stiff. However, most episodes resolve spontaneously within a few minutes of rest.
Episodes can be triggered by various factors such as exercise, excitement, swimming, stress, and extreme temperatures. The severity and frequency of the episodes can vary and are more commonly observed in the summer. Fortunately, as affected dogs age, the severity and frequency of the episodes tend to stabilize or decrease.
Avoiding activities that may trigger the episodes can be beneficial for managing this condition.
cost 48.00
more information is available on the following link:
Paradoxical Pseudomyotonia (PP)
|
new
X-Linked Retinal Dysplasia (RD / TRD)
Retinal dysplasia (RD) is an inherited disease caused by abnormal development of the retina, which is the layer of cells at the back of the eye, resulting in vision impairment from the time the eyes first open.
In Cocker Spaniels, this form of RD presents as total retinal detachment and severe visual deficits by 6 weeks of age. It is caused by a genetic mutation in the NDP gene, which is located on the X chromosome. Consequently, male dogs will be affected if they carry one copy of the defective gene.
cost 48.00
more information is available on the following link:
X-Linked Retinal Dysplasia (RD / TRD)
|
new
Bunny Hopping Syndrome(BHS1)
Bunny Hopping Syndrome (BHS1) in Weimaraners is an autosomal recessive movement disorder caused by a neurological developmental defect. In mammals, each half of the brain controls the opposite side of the body; for instance, nerve fibers from the left side of the brain innervate the right side of the body, and vice versa. For proper movement control, nerve fibers in the spinal cord must travel on only one side of the body. However, in dogs with BHS1, this orderly arrangement is disrupted, causing nerve strands in the spinal cord to switch sides.
As a result, affected dogs cannot control their left or right hind leg independently and instead hop with both legs simultaneously. This distinctive gait resembles that of a bunny, giving the condition its name, Bunny Hopping Syndrome. Unlike shaking puppy syndrome in Weimaraners, the gait of puppies affected by BHS1 does not improve over time, typically leading to euthanasia due to the lack of a cure.
more information is available on the following link:
cost 48.00
Bunny Hopping Syndrome
|
new
Dilated Cardiomyopathy (DCM)
Dilated cardiomyopathy (DCM) is a condition affecting the heart muscle, characterized by the thickening, dilation, and weakening of the left ventricle, the heart's main pumping chamber.
This impairment hinders the heart's ability to effectively circulate blood throughout the body. In the Nova Scotia Duck Tolling Retriever breed, DCM has been linked to an autosomal recessive variant in the LMNA gene. The disease manifests as sudden, paroxysmal accelerated heart activity (paroxysmal ventricular tachycardia).
Additional symptoms may include malformation (dysplasia) of the mitral valve, which is located between the left atrium and left ventricle, and tissue changes in the heart muscle (myocardial fibrosis). The onset age of DCM varies, and the disease can lead to sudden death at a young age, typically between 10 to 15 months.
cost 48.00
more information is available on the following link:
Dilated Cardiomyopathy (DCM)
|
new
Cleft Lip, Palate and Syndactyly (CLPS)
Cleft lip, palate, and syndactyly (CLPS) is a genetic condition that has been exclusively identified in Nova Scotia Duck Tolling Retrievers. There are two distinct genetic variants responsible for cleft palate in this breed: one in the CP1 gene and another in the ADAMTS20 gene, which also causes similar symptoms in humans. Both variants are inherited in an autosomal recessive manner.
CLPS is a congenital malformation where affected puppies are born with a hole in their palate, leading to difficulties in suckling and an increased risk of aspiration pneumonia due to milk entering the lungs. Additionally, some affected puppies may exhibit a shortened lower jaw (brachygnathia). In cases involving the ADAMTS20 variant, the condition may also manifest as abnormal crowding of the middle toes (syndactyly)
The test at Laboklin now detects two variants: CLPS and CP1. Both variants can cause Cleft Lip / Palate and Syndactyly. The price adjusted accordingly.
cost 66.00
more information is available on the following link:
Cleft Lip / Palate and Syndactyly (CLPS, CP1)
|
new
New study on DAMS
Dyserythropoietic Anaemia and Myopathy (DAMS), a recently discovered hereditary disease in English Springer Spaniels and other dogs, causes anaemia, muscle wasting and severe movement problems due to defective blood and muscle cells. The disease is inherited as an autosomal recessive trait, meaning that an affected dog has received one copy of the mutated gene from each parent.
A study investigated the prevalence of the mutation in Europe, analysing over 800 dog samples.
The mutated gene was found in a surprisingly high number of dogs, with an allele frequency of 9.7%. Centralised breeding programmes with appropriate genotyping could help to reduce the spread of the mutation and puppy mortality. Laboklin was centrally involved in this study. You can read the full paper here: www.mdpi.com
cost 48.00
more information is available on the following link:
Dyserythropoietic Anemia and Myopathy Syndrome (DAMS)
|
new
Primary Immunodeficiency Type 2 (PIP2)
Affected animals show an increased susceptibility to respiratory pathogens and display symptoms of refractory pneumonia.
In the medical history of the affected dogs, infections with gastrointestinal parasites and chronic diarrhoea as well as skin diseases and abscess formation were documented.
cost 48.00
more information is available on the following link:
Primary Immunodeficiency type 2 (PIPS2)
|
new
Multiocular Defect
Defects (MOD) typically suffer from cataracts, which in advanced stages lead to loss of vision.
Other clinical signs may include: small lens (microphakia), splitting of the lens (coloboma), enlarged eyeball (macrophthalmia), retinal folds and detachment, vitreous disease (vitreopathy) and secondary glaucoma.
The age at diagnosis is variable (0.5-10 years), with an average age of about 2 years.
cost 48.00
more information is available on the following link:
Multiocular Defect in Old English Sheepdog
|
new
Polycystic Kidney Disease (PKD2)
A genetic variant responsible for causing polycystic kidney disease (PKD2) has been identified in Siberian cats and Neva Masquerade breeds. PKD2 is a progressive and inherited condition characterized by the development of multiple fluid-filled cysts within the kidneys. Over time, these cysts grow in size and number, disrupting the normal kidney function and ultimately leading to kidney failure.
Affected cats may show symptoms such as increased thirst and urination, weight loss, decreased appetite, and lethargy. Early diagnosis through genetic testing is crucial for managing the disease and implementing appropriate care strategies to prolong the cat's quality of life.
The genetic test for PKD2 can help breeders identify carriers of the variant and make informed breeding decisions, reducing the prevalence of this condition in future generations. Regular veterinary check-ups and monitoring can also aid in managing the disease effectively.
cost 48.00
more information is available on the following link:
Polycystic Kidney Disease 2 (PKD2)
|
new
Startle Disease
Startle disease, also known as hyperekplexia, is a hereditary neurodegenerative disorder linked to impaired transport of the neurotransmitter glycine. Various genetic variants are known to cause startle disease in different dog breeds.
In Old English Sheepdogs, a variant in the SLC6A5 gene has been identified as the cause of Startle Disease. Affected puppies exhibit clinical symptoms at a very young age, such as pronounced stiffness of the leg muscles and muscle tremors in response to acoustic or tactile stimuli. These puppies are unable to stand or walk and display a rigid, extended posture in all four limbs. Symptoms tend to lessen when the dogs are relaxed or asleep but intensify with exercise. Cyanosis may also occur during suckling.
Unfortunately, affected puppies often need to be euthanized.
cost 66.00
more information is available on the following link:
Startle Disease in Old English Sheepdog
|
new
Microphthalmia
A genetic variant in the DNAJC21 gene has been identified in Portuguese Water Dogs, which causes microphthalmia with haematopoietic defects. Affected dogs exhibit a unilateral or bilateral reduction in eye size, often to less than 50% of the normal size. Additionally, they may present with various ocular defects, such as cataracts, corneal dystrophy, reduction or absence of the crystalline lens (microphakia/aphakia), glaucoma, retinal lesions, and persistent pupillary membranes.
Other associated symptoms may include changes in tooth enamel, growth retardation, and haematological abnormalities, such as low platelet counts (thrombocytopenia) and low red blood cell counts (anaemia).
As the affected dogs age, their red blood cell count and haematocrit may improve, while the platelet count typically remains low.
cost 48.00
more information is available on the following link:
Microphthalmia
|
new
Congenital Hypothyroidism (CH)
A genetic variant in the thyroid peroxidase (TPO) gene is responsible for primary congenital hypothyroidism in cats.
Affected cats exhibit disproportionate dwarfism, with growth retardation varying in visibility. Some cats show obvious signs, while in others, it is less apparent. Additional symptoms may include goitre characterized by the bilateral enlargement of both thyroid lobes, mental sluggishness, constipation, and delayed tooth eruption.
Serum total T4 concentrations in these cats are typically low to low-normal, while TSH concentrations are abnormally high.
Congenital hypothyroidism is generally diagnosed at an early age.
cost 48.00
more information is available on the following link:
Congenital Hypothyroidism (CH)
|
new
Muscular Dystrophy (MD)
Muscular dystrophy (MD) is an inherited condition characterized by slowly progressive muscle atrophy.
In the American Staffordshire Terrier breed, a variant in the COL6A3 gene has been identified as the cause of congenital muscular dystrophy.
Symptoms, including progressive gait problems and joint contractures, begin to appear around 6 months of age. Affected dogs exhibit widespread muscle atrophy and multifocal joint contractures, resulting in limited range of motion and significant thickening of the elbow and knee joints. Additionally, they show joint hyperlaxity (hyperextensible joints) in the distal limbs. Other symptoms include generalized weakness, difficulty in rising and walking, and tetraparesis, which is characterized by a stiff, jerky gait with short steps in all limbs but without obvious ataxia. Weak withdrawal reflexes are also observed in all limbs.
cost 48.00
more information is available on the following link:
Muscular Dystrophy (MD)
|
new
Double Coat
A dog's coat varies not only in the length and structure of individual hairs but also in whether it is a double coat (double layer) or single coat (single layer). Wolves, with their distinctive double coats consisting of two layers, are perfectly adapted to the environmental conditions of their habitat. This characteristic is also found primarily in working dog breeds such as sheepdogs and some hunting dog breeds.
Single coats are common in many short-haired breeds that are kept indoors or as family pets, such as watchdogs and various terrier breeds.
The two types of coats differ in function and hair structure. While all dogs have a primary or top coat that forms a protective top layer and contributes to the dog's color, the numerous fine hairs of the undercoat, found only in double-coated dogs, are very soft and serve as insulation.
Two genetic variants have been identified that correlate with double and single coats. The allele 'A' (ancestral), considered a wild-type variant, is found in both wolves and most dogs with double coats, while the recessive allele 'D' (derived) is predominantly found in homozygous form in dogs with single coats.
Genetic testing for these variants can be used in breeding programs to select dogs with the desired coat type and corresponding genotype. This is particularly important in double-coated breeds, where a single coat and the associated lack of an undercoat are considered faults according to the breed standard.
Preliminary statistics on the distribution of these variants suggest that other genetic factors may also play a role, as not all phenotypes correlate directly with the genotypes determined so far using the double coat test.
cost 48.00
more information is available on the following link:
Double Coat
|
new
Neuronal Ceroid Lipofuscinosis (NCL)
Neuronal ceroid lipofuscinosis (NCL) is characterized by the extensive accumulation of ceroid lipofuscin lipopigments in nerve cells and other tissues. This leads to cellular degeneration and eventual cell death, causing brain atrophy and progressive neurodegenerative symptoms.
A genetic variant of the MFSD8 gene associated with NCL (CLN7) has been identified in the Small Swiss Hound.
Symptoms typically begin to manifest around 12 months of age. Affected puppies exhibit progressive movement coordination disorders (ataxia), along with cognitive and visual impairment. Owners of affected dogs often report frequent collisions with obstacles, disorientation, difficulty climbing stairs, and sometimes increasingly agitated behavior.
cost 48.00
more information is available on the following link:
Neuronal Ceroid Lipofuscinosis (NCL)
|
Degenerative Myelopathy Risk Modifier DMRM / (Early-Onset)
The degenerative myelopathy (DM) risk modifier is a genetic variant that affects Pembroke Welsh Corgis.
This genetic mutation does not cause DM itself, but influences the onset of DM symptoms in dogs already affected by the common DM Exon 2 mutation in the SOD1 gene (DM/DM) . Symptoms in dogs that are genetically affected by DM Exon 2 typically manifest between 8 and 14 years of age, with the average age being around 11 years, however, in Pembroke Welsh Corgi, the presence of the DM risk modifier mutation (SP110) reduces the age of onset at which the disease manifests to the first two years of its age . Conversely, if a dog does not have two copies of the DM Exon 2 mutation, the SP110 variant does not impact its health.
The Degenerative Myelopathy (DM) risk modifier (SP11) has been identified in other breeds, but its influence on the age of onset has only been validated in Pembroke Welsh Corgis.
cost 48.00
more information is available on the following link:
Degenerative Myelopathy Risk Modifier DMRM / (Early-Onset)
|
new
Hair Shaft Dystrophy (HSD)
A genetic variant for hair shaft dystrophy and alopecia can now be analysed.
Hair Shaft Dystrophy (HSD) is a genetic condition identified in Cavalier King Charles Spaniels. This condition leads to localized alopecia, characterized by hair loss in specific areas. The genetic variant responsible for HSD affects the structure of the hair shaft, causing it to be fragile and prone to breakage. As a result, affected dogs may exhibit patches of hair loss, particularly in areas where the hair is subjected to friction or grooming.
more information is
available on the following link:
Hair Shaft Dystrophy (HSD)
|
new
PRA with Neurodegeneration PRA-PCYT2
Progressive Retinal Atrophy with Neurodegeneration is an inherited syndromic disease affecting the Saarloos Wolfdog breed and comprising blindness and neurological deficits.
Clinical signs includes early adult onset retinal degeneration, ophthalmological findings are consistent with generalized progressive retinal atrophy (PRA), with first clinical signs being observable between 20 and 46 months of age. Symptoms include early adult-onset neurological deficits with gait abnormalities, hind limb weakness, tremors, ataxia, cognitive decline and behavioural changes such as aggression towards the owner.
Additionally, epileptic seizures were reported in some cases. Affected dogs often have to be euthanized because of progression of the neurological signs and impaired quality of life.
Histopathological examination of an affected dog demonstrated cataract, retinal degeneration, central and peripheral axonal degeneration, and severe astroglia hypertrophy and hyperplasia in the central nervous system.
cost 48.00
more information is available on the following link:
PRA with Neurodegeneration PRA-PCYT2
|
new
Dachshund DNA bundle 2
- Chondrodystrophy (CDDY with IVDD Risk) and Chondrodysplasia (CDPA)
- Brittle Bone Disease (Osteogenesis Imperfecta)
- Neuronal Ceroid Lipofuscinosis ( CL / NCL )
- Progressive Retinal Atrophy (cord1- PRA / crd4 PRA) KC* DNA testing scheme in Miniature Dachshunds
- Progressive Retinal Atrophy (crd PRA)
cost 138.00
each
more information is
available on the following link:
Dachshund DNA bundle 2
|
new
Continental Bulldog DNA bundle
- Cystinuria
- Robinow-like Syndrome ( DVL2 )
- Coat Colour: B Locus (bd, bc, bs) Brown Coat Colour
- Canine Multi-Focal Retinopathy (CMR 1/2/3)
cost 126.00
more information is available on the following link:
Continental Bulldog (Conti) DNA bundle
|
new
Great Dane DNA bundle
- Myelopathy / Degenerative Radiculomyelopathy) DM (Exon 2) / SOD1
- Ichthyosis (Congenital Ichthyosis / Great Dane Ichthyosis)
- Hereditary Myopathy / Centronuclear Myopathy (HMLR, CNM / IMGD) KC* DNA scheme in Great Dane
- Leukoencephalomyelopathy ( LEMP )
cost 138.00
more information is
available on the following link:
Great Dane DNA bundle
|
new
Bull Mastiff DNA bundle
- Degenerative Myelopathy / Degenerative Radiculomyelopathy) DM (Exon 2) / SOD1
- Canine Multi-Focal Retinopathy (CMR 1/2/3)
- Cystinuria
- Mitochondrial Fission Encephalopathy (MFE) / Familial Cerebellar Ataxia
- Progressive Retinal Atrophy (Dominant PRA)
cost 138.00
more information is
available on the following link:
Bull Mastiff DNA bundle
|
new
Manchester Terrier / English Toy Terrier DNA bundle
- Dilated cardiomyopathy ( DCM / JDCM)
- von Willebrand disease Type I (vWD I) KC* DNA scheme in Manchester Terrier
- Hereditary Xanthinuria Type II (XDH-MOCOS)
cost 126.00
more information is available on the following link:
Manchester Terrier DNA bundle
|
new
Samoyed DNA bundle
- Amelogenesis Imperfecta (AI) / Familial Enamel Hypoplasia (FEH)
- Hereditary Nephritis / Samoyed Hereditary Glomerulopathy KC* DNA scheme in Samoyed
- XL - PRA (Progressive retinal Atrophy) KC* DNA scheme in Samoyed
cost 132.00
more information is available on the following link:
Samoyed DNA bundle
|
new
Havanese DNA bundle
- Degenerative Myelopathy / Degenerative Radiculomyelopathy) DM (Exon 2) / SOD1
- Haemophilia A (factor VIII deficiency / F8 )
- Chondrodystrophy (CDDY with IVDD Risk) and Chondrodysplasia (CDPA)
- Macrothrombocytopenia ( MTC-D )
- Furnishings
cost 138.00
more information is
available on the following link:
Havanese DNA bundle
|
new
Coat (hair) Types / Textures DNA Bundle
- Coat (hair) Length I ( Long or Short Hair)
- Coat (hair) Length II ( Long or Short Hair)
- Curly Coat: c1 + c2
- Furnishings
cost 138.00
more information is
available on the following link:
Coat (hair) Types / Textures DNA Bundle
|
update
Poodle Pack 2 DNA bundle
The bundle now includes:
- Degenerative Myelopathy / Degenerative Radiculomyelopathy) DM (Exon 2) / SOD1
- Neonatal encephalopathy (NE / NEWS)
- Progressive Retinal Atrophy (prcd-PRA): (8094P / 8127) KC* DNA testing scheme in Miniature, Standard and Toy Poodles
- Progressive retinal atrophy ( rcd4-PRA) / LOPRA KC* DNA testing scheme in , Standard and Toy Poodles
- von Willebrand disease Type I (vWD I) KC* DNA testing scheme in Standard Poodle
- Coat Colours: A-Locus Agouti ( fawn, sable, black and tan/tricolor, recessive black)
- Coat Colour: B Locus (bd, bc, bs) Brown Coat Colour
- Coat Colours: D-Locus D1 ( Dilution / Dilute )
- Coat Colours: E-Locus E1 (yellow, lemon, red, cream and appricot)
- Coat Colours: K- Locus (KB and ky)
- Coat Colour: I - Locus (phaeomelanin intensity)added at no extra cost
- Coat Colour: S-locus (piebald, spotted white)* added at no extra cost
cost 180.00
more information is
available on the following link:
Poodle Pack 2 DNA bundle
|
update
Vizsla DNA bundle
The bundle now includes:
- Degenerative Myelopathy / Degenerative Radiculomyelopathy) DM (Exon 2) / SOD1
- Exfoliative Cutaneous Lupus Rrythematosus ( ECLE ) / Lupoid Dermatosis
- Neonatal Cortical Cerebellar Abiotrophy (NCCD)
- Coat (hair) Length I ( Long or Short Hair)
- Furnishings
- Hyperuricosuria / Urate Stones (HUU, SLC) KC* DNA scheme in Hungarian Wirehaired Vizsla (Vizslak)added at no extra cost
cost 138.00
more information is
available on the following link:
Vizsla DNA bundle
|
update
English Springer Spaniel DNA bundle
The ESS DNA bundle has now been updated to include:
- Acral Mutilation Syndrome ( AMS )
KC* DNA scheme in English Springer Spaniel
- Familial Nephropathy (FN) / Hereditary Nephropathy
- Hypomyelination (Shaking Puppy Syndrome) SPS
- Fucosidosis
KC* DNA scheme in English Springer Spaniel
- PFK Deficiency (Phosphofructokinase deficiency / PFKD)
KC* DNA scheme in English Springer Spaniel
- Progressive Retinal Atrophy (cord1- PRA / crd4 PRA)
KC* DNA scheme in English Springer Spaniel
- Dyserythropoietic Anemia and Myopathy Syndrome (DAMS)
KC* DNA scheme in English Springer Spaniel
cost 138.00
more information is
available on the following link:
English Springer Spaniel DNA bundle
|
update
Cavalier King Charles Spaniel Pack B DNA bundle
The Cavalier King Charles Spaniel Pack B has been updated to include:
- Episodic Falling in Cavalier King Charles Spaniel (EF)
KC* DNA scheme in Cavalier King Charles Spaniel
- Dry Eye and Curly Coat syndrome (CCS)
KC* DNA scheme in Cavalier King Charles Spaniel
- MCAD deficiency ( MCADD )
- Myxomatous mitral valve disease (MMVD)
- Macrothrombocytopenia ( MTC-D )
cost 138.00
more information is
available on the following link:
Cavalier King Charles Spaniel Pack B DNA bundle
|
update
White Swiss Shepherd DNA bundle
The White Swiss Shepherd ( Berger Blanc Suisse ) DNA bundle has been updated to include:
- Degenerative Myelopathy / Degenerative Radiculomyelopathy) DM (Exon 2) / SOD1
- MDR1 Gene Varian / Ivermectin Sensitivity * (ABCB1)
- Dwarfism (Pituitary Dwarfism / Hypopituitarism)
- Coat (hair) Length I ( Long or Short Hair)
- Cerebellar Hypoplasia (CH)
cost 138.00
more information is
available on the following link:
White Swiss Shepherd DNA bundle
|
new Kennel Club DNA testing schemes
The following tests are now part of the Kennel Club DNA testing schemes:
- Degenerativ Myelopathy DM Exon 2 in Rhodesian Ridgeback
more ..
- GM1-Gangliosidosis in Shiba Inu more ..
- GM2 Gangliosidosis Variant 0 (Sandhoff Disease) in Shiba Inu more ..
|
testing is easy
To test a dog, we need a DNA sample, which can be collected using simple mouth swabs or a blood sample collected by the vet. Both buccal swabs and blood tubes are available from us free of charge. Samples can be sent to our Manchester address below.
Turnaround 2-3 weeks or sooner for most DNA test.
Request Sample Materials
LABOKLIN is a DIN EN ISO / IEC 17025 Accredited laboratory. This means that all our tests are performed according to the reliable accreditation standards and verified procedures.
|
Stay up to date with all the news and special offers by visiting our facebook page
|
|
|
|
|
If
you
no
longer
wish
to
receive
mail
from
us,
you
can unsubscribe
LABOKLIN
(UK),
Unit 20, Wheel Forge Way, Trafford Park
Manchester,
Greater
Manchester,
M17
1EH,
United
Kingdom
|
|
|
|
|
|
|
